summary: This study found that a protein which is part of the sodium channel involved in nerve conduction is disrupted in MS during demyelination and seems to be active during remyelination too, helping our understanding of mechanisms of damage and repair.

authors: Howell OW, Palser A, Polito A, Melrose S, Zonta B, Scheiermann C, Vora AJ, Brophy PJ, Reynolds R.

source: Brain. 2006 Dec;129(Pt 12):3173-85. Epub 2006 Oct 14.

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category: Pathology

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